Hundreds of studies since NASA’s groundbreaking research in 1996 and continuing today at venerable institutions like Harvard, Mass General and Stanford have show that Photobiomodulation affects the cells of the body in a number of beneficial ways with positive side effects. These effects speed healing, soothe sore muscles, ease chronic pain, relieve stiffness, and increase circulation. Light exposure can do this by improving mitochondrial efficiency and protecting against inflammation. Light therapy has also been shown to help mitochondria make ATP faster.
Each wavelength or color seems to have its own level of effectiveness and what we call a ‘healing signature’. Varying the wavelengths of light creates benefits that work in synergy with each other to reduce the toxic threshold and inflammation within the cells, increase cellular energy, circulation and thus increase cell life. When you can heal the cells without harmful side effects, the benefits cascade down into the tissues, organs and system, benefiting the entire body.
The key cellular mechanisms of LED Light Therapy include the following:
(1) reduction in inflammation (the leading cause of mortality and morbidity diseases)
(2) Increases the size and density of energy producing mitochondria (mitochondrial dysfunction is a significant sign of age related decline and represents the current theory of why we age).
(3) detoxify and nourish the cells
We invite you to take note of #1 and #2 above. Chronic, low level inflammation is the leading cause of disease, and mitochondrial dysfunction is a leading cause of age related decline. LED Light Therapy has been shown to reduce inflammation and restore mitochondrial function (creates ‘giant mitochondria’).
THIS SAYS IT ALL
Read what researchers from Mass General, Harvard and Boston University conclude about LED Light Therapy.
2.8. Downstream cellular response
There have been a large number of both animal model and clinical studies that demonstrated highly beneficial LLLT effects on a variety of diseases, injuries, and has been widely used in both chronic and acute conditions (see Figure 7). LLLT may enhance neovascularisation, promote angiogenesis and increase collagen synthesis to promote healing of acute (Hopkins et al. 2004) and chronic wounds (Yu et al. 1997). LLLT provided acceleration of cutaneous wound healing in rats with a biphasic dose response favoring lower doses (Corazza et al. 2007). LLLT can also stimulate healing of deeper structures such as nerves (Gigo-Benato et al.2004), tendons (Fillipin et al. 2005), cartilage (Morrone et al. 2000), bones (Weberet al. 2006) and even internal organs (Shao et al. 2005). LLLT can reduce pain (Bjordal et al. 2006a), inflammation (Bjordal et al. 2006b) and swelling (Carati et al. 2003) caused by injuries, degenerative diseases or autoimmune diseases. Oron reported beneficial effect of LLLT on repair processes after injury or ischemia in skeletal and heart muscles in multiple animal models in vivo (Ad and Oron 2001; Oron et al. 2001a; Oron et al. 2001b; Yaakobi et al. 2001). LLLT has been used to mitigate damage after strokes (in both animals (Lapchak et al. 2008) and humans (Lampl et al. 2007)), after traumatic brain injury (Oron et al. 2007) and after spinal cord injury (Wu et al. 2009).
Beneficial tissue effects of LLLT can include almost all the tissues and organs of the body.